全球指纹与皮纹科学研究文献整理 (Part VII)
109. This writer has four very well formed and centrally located, perhaps even five, peacock’s eyes, including two on the little fingers, one on the right ring finger, one on the left middle finger and one in a reverse loop on the left thumb. He is right handed. All of the eyes form as pockets of ulnar loops and are part of the reason why he studies the palm. He has also walked away from several serious auto accidents and other traumas.
112. The Hand As A Mirror Of Systemic Disease, Theodore J. Berry, M.D., F.A.C.P., F. A. Davis Company, Publishers, Philadelphia, 1963, for early development of dermatoglyphic patterns see also Dermatoglyphics in Medical Disorders, by B. Schaumann and M. Alter (1976) New York: Springer-Verlag, pp. 187 – 189.
116. William J. Babler, Prenatal Communalities in Epidermal Ridge Development, pp. 54-68 in Trends in Dermatoglyphic Research, edited by Norris M. Durham and Chris C. Plato, Kluwer Academic Publishers, Dordrecht/London/Boston ? 1990. (Vol 1, Studies in Human Biology)
122. William J. Babler, Prenatal Communalities in Epidermal Ridge Development, supra n. 64, p. 67 referring to S. M. Garn, W. J. Babler & A. R. Burdi, Prenatal origins of brachymesophalangia-5. Am. J. Phys. Anthrop. (1976) 91: 147-173
126. Ibid. They confirm that the volar pads on the index and middle fingers are visible in the second month. They relate the symmetry or asymmetry of the pads and their development to whether whorls, loops or arches may be expected, with symmetrical = whorls, asymmetrical = loops and weak pad development = arches. They indicate that the pad shape is genetically set though environmentally modified. When crown rump length reaches about 90 mm (4th month) the first distinct, sharply delineated fold like proliferations appear in the epidermis and these are later perceived as glandular folds. These glandular folds bear a close spacial relationship with the distribution of capillary-neurite pairs. On the basis of the pattern of these glandular folds, they predicted that the forces of the growth pressure of these folds, the trajectory of the system of the epidermis and the glandular ducts would determine the highly arranged surface pattern of the papillary ridges. The final expression of genetic information in the form of surface patterns would be modified by environmental influences. Glandular folds , proliferations of cells from the epidermis that make their way into the mesenchyme (dermis) and form from the lateral distal borders of the distal phalanx to the medial proximal part of that phalanx and forming a Horshoe-shaped border on the fingertip. Folds continue to be formed at the periphery until the pad surface is entirely covered. This process occurs on the proximal phalanges beginning in the fifth month. During the fifth month the sweat glands set and the glandular ducts reach the surface in the sixth month. During the later course of development of the glandular folds, the volar pads become increasingly less prominent while the connective tissue becomes richer in collagen and denser. Secondary furrow fold formation is seen in the sixth and seventh month but it has either slight or no effect on the formation of the papillary ridge pattern. The papillary ridges on the surface skin molded by the glandular fold cell proliferation after the formation of glandular folds, sweat gland secretion and keratinisation has begun, after the sixth month.
The authors speculate that three factors may possibly accomplish the transfer of the deep patterns to the skin surface: 1) proliferation pressure from the increased mitotic rate of the basal cell layer; 2) stabilization of the sweat gland secretion ducts at regular intervals on the surface; and, 3) by some counteracting force as a result of the first two forces, exerted by the tonofilament system, the system of fine filaments in the cytoplasm of each cell that function as supportive elements within the cytoskeleton and form the main precursors of keratin in the epithelium. They conclude that the pattern of the papillary ridges is set after the development of the glandular folds, after the forth month.
128. Richard E. Behrman, M.D., Robert M. Kleigman, M.D. and Ann . Arvin, M.D., Nelson Textbook of Pediatrics, 15th Edition ? 1996, p. 34, W. B. Saunders Company, division of Harcourt Braced & Company, Philadelphia/London/Toronto/ Montreal/ Sidney/Tokyo.
131. Sumiko Kimura, Blanka A. Schaumann, Chris C. Plato and Tadashi Kitagawa, Developmental Aspects of Human Palmar, Plantar, and Digital Flexion Creases, in Trends in Dermatoglyphic Research, edited by Norris M. Durham and Chris C. Plato, Kluwer Academic Publishers, Dordrecht/London/Boston ? 1990. (Vol 1, Studies in Human Biology), p. 84.
147. W. Hirsch, Dermatoglyphics and Creases in Their Relationship to Clinical syndromes: A Diagnostic Criterion. pp. 263-282 in Jamshed Mavalwala, Editor, Dermatoglyphics, An International Perspective, 1978, Moulton Publishers, The Hague/Paris.
148. Dermatoglyphics in Medical Disorders, Blanka Schaumann, Milton Alter, supra., pp. 146-172. Danuta Z. Loesch, Quantitative Dermatoglyphics, Classification, Genetics, and Pathology, ?1983, pp. 220-289 Oxford Monographs on Medical Genetics, Hartnoll Print, Bodmin, Cornwall, UK.
150. Theodore Millon & Roger D. Davis, The Millon Clinical Multiaxial Inventroy-III (MCMI-III), in Major Psychological Assessment Instruments 2nd Ed. Pp. 108-147, Charles S. Newmark Ed. Allyn & Bacon, ? 1985-1996